[1] Huang PN, Shih SR.Update on enterovirus 71 infection[J]. Curr Opin Virol, 2014, 5: 98-104. [2] Zhang Y, Zhu Z, Yang W, et al.An emerging recombinant human enterovirus 71 responsible for the 2008 outbreak of hand foot and mouth disease in Fuyang city of China[J]. Virol J, 2010, 7: 94. [3] Wang H, Li Y.Recent Progress on Functional Genomics Research of Enterovirus 71[J]. Virol Sin, 2019, 34(1): 9-21. [4] 王宏,谢广成,段招军. 小RNA病毒3C蛋白酶研究进展[J]. 病毒学报,2014,30(5):579-586. [5] 谢广成,段招军. EV71的感染和抗病毒天然免疫应答[J]. 病毒学报,2016,32(4):501-508. [6] Wong WR, Chen YY, Yang SM, et al.Phosphorylation of PI3K/Akt and MAPK/ERK in an early entry step of enterovirus 71[J]. Life Sci, 2005, 78(1): 82-90. [7] Zhang H, Li F, Pan Z, et al.Activation of PI3K/Akt pathway limits JNK-mediated apoptosis during EV71 infection[J]. Virus Res, 2014, 192: 74-84. [8] Peng H, Shi M, Zhang L, et al.Activation of JNK1/2 and p38 MAPK signaling pathways promotes enterovirus 71 infection in immature dendritic cells[J]. BMC Microbiol, 2014, 14: 147. [9] Zhang H, Cong H, Song L, et al.The nuclear protein Sam68 is redistributed to the cytoplasm and is involved in PI3K/Akt activation during EV71 infection[J]. Virus Res, 2014, 180: 1-11. [10] 李丹,苏萌,孙萍萍,等. 基于Label-free定量蛋白组学揭示肠道病毒A71型感染致人扁桃体上皮细胞蛋白质组的改变[J]. 病毒学报,2020,36(5):778-786. [11] Li D, Su M, Sun PP, et al.Global profiling of the alternative splicing landscape reveals transcriptomic diversity during the early phase of enterovirus 71 infection[J]. Virology, 2020, 548: 213-225. [12] Hawkins PT, Anderson KE, Davidson K, et al.Signalling through Class I PI3Ks in mammalian cells[J]. Biochem Soc Trans, 2006, 34(Pt 5): 647-662. [13] Vanhaesebroeck B, Ali K, Bilancio A, et al.Signalling by PI3K isoforms: insights from gene-targeted mice[J]. Trends Biochem Sci, 2005, 30(4): 194-204. [14] Xie GC, Guo NJ, Grenman R, et al.Susceptibility of human tonsillar epithelial cells to enterovirus 71 with normal cytokine response[J]. Virology, 2016, 494: 108-118. [15] Yuan J, Shen L, Wu J, et al.Enterovirus A71 Proteins: Structure and Function[J]. Front Microbiol, 2018, 9: 286. [16] Plevka P, Perera R, Cardosa J, et al.Crystal structure of human enterovirus 71[J]. Science, 2012, 336(6086): 1274. [17] Chen P, Song Z, Qi Y, et al.Molecular determinants of enterovirus 71 viral entry: cleft around GLN-172 on VP1 protein interacts with variable region on scavenge receptor B 2[J]. J Biol Chem, 2012, 287(9): 6406-6420. [18] Lin YL, Yu CI, Hu YC, et al.Enterovirus type 71 neutralizing antibodies in the serum of macaque monkeys immunized with EV71 virus-like particles[J]. Vaccine, 2012, 30(7): 1305-1312. [19] Kiener TK, Jia Q, Lim XF, et al.Characterization and specificity of the linear epitope of the enterovirus 71 VP2 protein[J]. Virol J, 2012, 9: 55. [20] Kiener TK, Jia Q, Meng T, et al.A novel universal neutralizing monoclonal antibody against enterovirus 71 that targets the highly conserved "knob" region of VP3 protein[J]. PLoS Negl Trop Dis, 2014, 8(5): e2895. [21] Shingler KL, Yoder JL, Carnegie MS, et al.The enterovirus 71 A-particle forms a gateway to allow genome release: a cryoEM study of picornavirus uncoating[J]. PLoS Pathog, 2013, 9(3): e1003240. [22] Pathinayake PS, Hsu AC, Wark PA.Innate Immunity and Immune Evasion by Enterovirus 71[J]. Viruses, 2015, 7(12): 6613-6630. [23] Yu J, Zhang L, Ren P, et al.Enterovirus 71 mediates cell cycle arrest in S phase through non-structural protein 3D[J]. Cell Cycle, 2015, 14(3): 425-436. [24] Shin YK, Liu Q, Tikoo SK, et al.Influenza A virus NS1 protein activates the phosphatidylinositol 3-kinase (PI3K)/Akt pathway by direct interaction with the p85 subunit of PI3K[J]. J Gen Virol, 2007, 88(Pt 1): 13-18. [25] Mazzon M, Castro C, Thaa B, et al.Alphavirus-induced hyperactivation of PI3K/AKT directs pro-viral metabolic changes[J]. PLoS Pathog, 2018, 14(1): e1006835. [26] Cuadrado A, Nebreda AR.Mechanisms and functions of p38 MAPK signalling[J]. Biochem J, 2010, 429(3): 403-417. [27] Sugasti-Salazar M, Llamas-Gonzalez YY, Campos D, et al.Inhibition of p38 Mitogen-Activated Protein Kinase Impairs Mayaro Virus Replication in Human Dermal Fibroblasts and HeLa Cells[J]. Viruses, 2021, 13(6): 1156. [28] Zhan Y, Yu S, Yang S, et al.Newcastle Disease virus infection activates PI3K/Akt/mTOR and p38 MAPK/Mnk1 pathways to benefit viral mRNA translation via interaction of the viral NP protein and host eIF4E[J]. PLoS Pathog, 2020, 16(6): e1008610. [29] Cheng Y, Sun F, Wang L, et al.Virus-induced p38 MAPK activation facilitates viral infection[J]. Theranostics, 2020, 10(26): 12223-12240. [30] Yu T, Yang Q, Tian F, et al.Glycometabolism regulates hepatitis C virus release[J]. PLoS Pathog, 2021, 17(7): e1009746. |